Clinical Tool

Disorder Comparison Table

Anxiety Disorder Comparison Table

Quick Reference Tool | PsychHQ Source: Module 5, Disorder-Specific Deep Dives (Sections 1–8) | Last Updated: March 2026


At a Glance: Eight Anxiety Disorders Compared

Disorder Core Cognitive Driver Typical Onset Course First-Line Psychotherapy First-Line Pharmacotherapy Key Distinguishing Feature
GAD Intolerance of uncertainty; worry as avoidance of emotional imagery Adolescence to early adulthood Chronic, relapsing-remitting CBT matched to model: worry exposure (Borkovec), behavioral experiments for uncertainty (Dugas), or MCT [Metacognitive Therapy] (Wells) SSRI/SNRI (2025 Cochrane NNT [number needed to treat] = 7; NNH [number needed to harm — how many patients treated before one discontinues due to side effects] = 17) Worry shifts across domains; neither GAD nor MDD is inherently primary
Social anxiety Catastrophic fear of negative evaluation; self-focused attention Mean age ~13 Chronic if untreated; 36% wait 10+ years to seek help Individual Clark & Wells CBT (behavioral experiments, video feedback, safety behavior cessation) SSRI (NNT ~4.2–4.7); phenelzine g = 1.14 but reserved for treatment-resistant cases due to dietary and drug interaction constraints Performance inhibition, not skill deficit; 90-minute sessions outperform 50-minute
Panic disorder Catastrophic misinterpretation of benign body sensations (Clark, 1986) Late adolescence to mid-30s Remission 75% uncomplicated / 67% with agoraphobia; relapse 12% / 21% CBT with interoceptive exposure (IE — deliberately inducing feared body sensations) SSRI (start at sub-therapeutic dose, titrate slowly — activation syndrome is almost invariably catastrophically misinterpreted in this population) IE is the highest-efficacy component; including relaxation during exposure associated with reduced efficacy in dismantling data
Agoraphobia Fear of situations where escape is difficult or help unavailable Often within 12 months of first panic attack; >80% develop agoraphobia in the first year Chronic without treatment Systematic in-vivo situational exposure; telehealth CBT produces equivalent large effects SSRI (same algorithm as panic) Now independent from panic in DSM-5; ~25% of treatment-seeking youth lose eligibility under more stringent DSM-5 criteria
Specific phobias Conditioned or prepared fear of a specific stimulus Median age ~8 Persistent; only 23.1% ever receive treatment In-vivo exposure; OST (one session, up to 3 hours; pre-to-post g = 1.15 across subtypes [Ollendick & Davis, 2013]; ASPECT trial N = 268: non-inferior to multi-session CBT [Wright et al., 2023]) Not first-line; propranolol or short-course BZD for single unavoidable events only Most prevalent, least treated; in 72.6% of comorbid cases, the phobia preceded the onset of later disorders
BII phobia Same as specific phobia + vasovagal syncope risk Childhood to early adolescence Persistent Exposure + Applied Tension (tensing large muscles for 10–15 sec to prevent parasympathetic rebound) Not first-line Diphasic (two-phase) autonomic response: sympathetic surge → parasympathetic collapse → vasovagal syncope (fainting from sudden blood pressure drop); unique genetic architecture (81% of genetic variance from a BII-specific factor)
Separation anxiety Anticipatory dread of separation from attachment figure Bimodal: early childhood and late teens/20s 36.1% persist to adulthood; 77.5% of adult cases onset in adulthood Parent-based CBT with accommodation reduction (children); SPACE (parent-only, non-inferior to child CBT); adapted exposure hierarchies (adults) SSRI (CAMS: combination 80.7% response pooled across SAD/GAD/social phobia) Person-based avoidance (goes away when the attachment figure is present); BNST (bed nucleus of the stria terminalis — the "extended amygdala" that processes sustained, anticipatory threats)-mediated sustained threat processing
Selective mutism Anxiety-driven freeze of speech production in specific social contexts Ages 3–6 Persistent without intervention School-based behavioral protocols: IBT-SM (75% response in pilot RCT), PCIT-SM, sliding-in technique Limited evidence; fluoxetine in one small RCT; not first-line Context-dependent: fluent at home, mute at school; the "rescue cycle" in schools maintains the silence

Disorder-Specific Clinical Notes

GAD: Model-Matching Matters

Three maintenance models drive different treatment approaches. Borkovec: worry functions as avoidance of emotional imagery — treatment uses sustained imaginal exposure to worst-case scenarios (25–30 min without verbal rumination; d = 1.81 vs non-therapy controls, 57% full recovery at 12 months; Hanrahan et al., 2013). Dugas: intolerance of uncertainty is the catalyst — treatment uses behavioral experiments inducing uncertainty (going to a restaurant without checking reviews, sending an email with a deliberate typo). Wells: metacognitive beliefs about worry maintain the cycle ("I can't stop worrying," "this will drive me crazy") — treatment uses Detached Mindfulness and worry postponement (57.1% recovery at 9 years vs 37.5% for CBT; Solem et al., 2021 — single research group, small samples).

Assessment shortcut: Does the patient fear what they're worrying about (Borkovec/Dugas) or fear the worry itself (Wells)?

Buspirone niche: 5-HT1A partial agonist (a serotonin receptor that regulates anxiety without affecting GABA receptors used by benzodiazepines); no sedation, no dependence risk. Best for older adults at fall risk, patients with substance use histories, or SSRI partial responders needing augmentation. Onset 2–4 weeks — entirely ineffective as PRN.

Social Anxiety: Behavioral Experiments, Not Just Habituation

The Clark & Wells protocol has the largest point estimate in the anxiety psychotherapy literature (SMD = −1.56 in a sub-model analysis; Mayo-Wilson et al., 2014, 101 RCTs, 13,164 pts — this is a sub-model estimate without separate CIs; the aggregate individual CBT class effect is −1.19, 95% CrI [credible interval — the Bayesian equivalent of a confidence interval]: −1.56 to −0.81). The key insight: behavioral experiments test catastrophic predictions rather than waiting for fear to habituate. A patient who believes "if I sweat visibly, people will laugh" might spray water on their underarms before presenting.

Session length matters: English trials using 90-minute sessions averaged 6 behavioral experiments per course; German/Swedish trials using 50-minute sessions averaged 1.7. This is observational, not randomized, but it means that standard 50-minute hours may be a structural barrier to delivering the active ingredient.

Social skills training additive benefit is inconsistent. Most patients have adequate skills that are suppressed by anxiety — when the anxiety is treated, the skills emerge. Exception: comorbid Autism Spectrum Disorder, where genuine skill deficits may require separate intervention.

Panic: The Suffocation False Alarm and Activation Syndrome

Clark's model explains expected panic; Klein's suffocation false alarm (SFA) theory explains spontaneous and nocturnal attacks — the brain's CO2 monitor is set too sensitively, triggering a suffocation alarm from normal breathing variation.

Alprazolam publication bias: Turner & Ahn-Horst (2023) found that of 5 phase 2/3 trials for alprazolam XR, the FDA deemed only 1 (20%) genuinely positive. Two negative trials were suppressed; two published with post-hoc analyses conveying false positive outcomes. True effect size g = 0.33 vs published g = 0.47 (42% inflation).

Prescriber alert — activation syndrome: SSRIs frequently cause an initial spike in agitation during weeks 1–2. In panic patients specifically, this is catastrophically misinterpreted as the medication "making things worse." Pre-framing this for the patient before it occurs reduces early dropout.

Specific Phobias: One Session Can Be Enough

OST (Ost, 1989) consolidates the entire treatment into a single session of up to 3 hours: graduated in-vivo exposure, participant modeling (therapist touches the spider first), behavioral experiments, and reinforcement. Response rates ≥76%; pre-to-post effect size g = 1.15 across phobia subtypes and age groups (Ollendick & Davis, 2013). At 4-year follow-up, 90% of adults remain improved. The ASPECT trial (N = 268, ages 7–16) confirmed non-inferiority to multi-session CBT (Wright et al., 2023).

Phobias are developmental sentinels: in 72.6% of comorbid cases, the specific phobia preceded all other disorders (Wardenaar et al., 2017). Treating phobias early may interrupt downstream cascades.

Separation Anxiety: Not Just for Kids

Adult-onset SAD is more common than childhood SAD (lifetime prevalence 6.6% vs 4.1%; Shear et al., 2006). The DSM-5 eliminated the requirement for onset before age 18. Adult presentations: excessive worry about a partner's safety, "helicopter parenting," inability to tolerate partner's travel, sleep disruption when separated.

Differential from agoraphobia: Agoraphobia is place-based (fear of situations where escape is hard). SAD is person-based (fear of separation from the attachment figure). The test: an adult with agoraphobia still panics in a stadium with their spouse present. An adult with SAD is fine in that stadium if the spouse is there.

Selective Mutism: Break the Rescue Cycle

The silence is involuntary — an anxiety-driven freeze response, not defiance. The school "rescue cycle" maintains it: child freezes → teacher answers for them → child's anxiety drops → silence is reinforced → teacher's discomfort is relieved → rescuing is reinforced.

Treatment reversal: forced-choice questions (not open-ended demands), 5-second wait time resisting the rescue impulse, labeled praise for any communicative approximation. Defocused communication (side-by-side positioning, parallel activity, no direct eye contact) reduces the interpersonal pressure that triggers the freeze.

Warning: Punishing silence does not treat SM. Reward charts demanding speech without managing the underlying terror are counterproductive. SM requires the same graduated desensitization approach as severe phobias.


Tricky Differentials

Overlap How to Distinguish
GAD vs. MDD 4 of 6 GAD somatic criteria are identical to MDD criteria; 59–70% shared heritability. But the temporal relationship is symmetrical (37% anxiety first, 32% depression first, ~30% concurrent; Moffitt et al., 2007). Neuroimaging classifiers can distinguish the two above chance (Hilbert et al., 2017). Treat whichever is more severe at presentation.
GAD vs. social anxiety GAD worry is diffuse and shifts across domains. Social anxiety worry is focused on evaluation by others. The PSWQ (Penn State Worry Questionnaire — a 16-item measure of pathological worry) cutoff of 61–65 reliably separates GAD from SAD.
Panic vs. medical emergency Panic is a diagnosis of exclusion in the acute moment. Standard workup: vitals, ECG, pulse oximetry, metabolic panel. Post-ruling-out, the treatment target is the catastrophic misinterpretation, not the sensations themselves.
Separation anxiety vs. agoraphobia SAD = person-based avoidance (calms when attachment figure present). Agoraphobia = place-based avoidance (persists regardless of companion).
Selective mutism vs. oppositional defiance SM children are typically compliant, eager to please, and highly verbal at home. The silence is context-specific and involuntary. ODD presents with active refusal, arguing, and rule-breaking across settings.
Specific phobia vs. normative fear Average child reports 2–5 fears at any time. Phobia = persistence (≥6 months), disproportionality to actual threat, and functional impairment. ~23% of common childhood fears meet clinical criteria (Muris et al., 2000).

Key References: Kopcalic et al. (2025, Cochrane — GAD NNT); Mayo-Wilson et al. (2014, Lancet Psychiatry — SAD NMA); Pompoli et al. (2018, Psychol Med — panic dismantling); Wright et al. (2023, JCPP — ASPECT/OST); Wardenaar et al. (2017, Psychol Med — phobia epidemiology); Shear et al. (2006, Am J Psychiatry — adult SAD); Lebowitz et al. (2020, JAACAP — SPACE); Bergman et al. (2013, BRT — IBT-SM); Barlow et al. (2017, JAMA Psychiatry — UP equivalence); Moffitt et al. (2007, Arch Gen Psychiatry — GAD-MDD temporal symmetry); Clark (1986, BRT — panic cognitive model); Turner & Ahn-Horst (2023, Psychol Med — alprazolam publication bias)