Drug Comparison

For educational purposes only — a decision-support tool, not a substitute for clinical judgment.

Side-by-side rubric across 96 psychiatric medications. Every rating traces to a verbatim primary-source quote — click any cell to see it.

How to read this tool
Rating scale
Favorable / lower than class baseline
± Minimal / equivocal
+ Low / uncommon
++ Moderate / common
+++ High / very common
++++ Very high / class-outlier
Frequency vs severity
F = frequency, S = severity. Each gets its own pill colored on the same traffic-light scale: greenblueyelloworangered. Click any cell for incidence percentages and NNH.
Evidence tier
A Network meta-analysis / RCT / FDA label
B Cohort / registry / pooled label data
C Expert review / textbook / case series
Sourcing
Click any cell to see the verbatim source quote and citation. Missing data shows n/a.
Data depth
++ Graded — frequency + severity, primary-source traces
+ FDA label — §6 frequency only (dashed border). Click for sub-types.
  Blank — not yet checked (not “absent”)
±++++++++++ABCF = frequency · S = severity · Dashed border = FDA label only · Click cell for details
⚠ Cross-class comparison (NRI vs alpha-2-agonist vs SNRI-ADHD) — class floors may not apply uniformly.
4 drugs selected — Atomoxetine, Guanfacine, Viloxazine, Clonidine(click to collapse)
4/4 selected
Atomoxetine
Strattera
Selective Norepinephrine Reuptake Inhibitor
FDA-approved indications
  • ADHD (adults; pediatric 6+)
Off-label uses
  • Anxiety disorders with ADHD
  • Binge eating disorder
Half-life5 hours
Guanfacine
Intuniv · Guanfacine extended-release
Alpha-2 Adrenergic Agonist
FDA-approved indications
  • ADHD — mono or adjunct to stimulants (children/adolescents 6–17 years; ER formulation)
Off-label uses
  • PTSD
  • Tic disorders
  • Anxiety disorders (pediatric)
Half-life17 hours
Viloxazine
QELBREE
Serotonin-Norepinephrine Modulating Agent
FDA-approved indications
  • Attention-deficit/hyperactivity disorder (adults; pediatric 6+)
Off-label uses
  • Anxiety disorders (with ADHD)
Half-life7 hours
Clonidine
Kapvay
Alpha-2 Adrenergic Agonist
FDA-approved indications
  • ADHD — mono or adjunct to stimulants (children/adolescents 6–17 years; ER formulation)
Off-label uses
  • PTSD nightmares
  • Tic disorders
  • Opioid withdrawal
Half-life12 to 16 hours
Next:Taper AtomoxetineTaper GuanfacineTaper ViloxazineTaper ClonidineSwitching Guide →
Decision GuideWhen to pick each / when to consider an alternative
Atomoxetine
Consider when
  • ADHD with comorbid substance use disorder — non-scheduled; no abuse potential; preferred when stimulant diversion risk is high
  • ADHD with comorbid anxiety — anxiolytic effect documented in trials; stimulants may worsen anxiety
  • 24-hour ADHD coverage needed — continuous NRI effect without wearing-off; covers morning routine through sleep onset
  • Tic disorder comorbidity — does not exacerbate tics; may improve ADHD symptoms without worsening Tourette's
  • +1 more
Consider an alternative when
  • Rapid ADHD symptom response needed — takes 4–6 weeks for full effect vs stimulant onset within hours
  • Hepatic impairment — CYP2D6 substrate; dose reduction required in hepatic impairment; rare hepatotoxicity reported
  • On potent CYP2D6 inhibitor (paroxetine, fluoxetine, bupropion) — levels increase 6–8× in PMs or with strong inhibitors
  • Suicidal ideation risk in children/adolescents — boxed warning for increased SI in pediatric patients (0.4% vs 0%)
  • +1 more
Guanfacine
Consider when
  • ADHD with comorbid tics or Tourette syndrome — FDA-approved ADHD (ER, ages 6–17); improves both ADHD and tic symptoms
  • Stimulant-induced insomnia or emotional lability — α2A agonism calms hyperarousal; useful adjunct to stimulants
  • ADHD with comorbid oppositional behavior — evidence for reducing oppositional/conduct symptoms beyond ADHD core
  • Hypertension comorbidity — antihypertensive mechanism provides dual benefit in ADHD patients with elevated BP
  • +1 more
Consider an alternative when
  • Hypotension or bradycardia risk — dose-dependent BP and HR reduction; syncope risk especially during titration
  • Abrupt discontinuation likely — rebound hypertension with sudden cessation; must taper over 3–7 days
  • Daytime sedation is problematic — somnolence 13–38% across doses; leading side effect; may impair school/work performance
  • Maximum ADHD efficacy needed — effect sizes smaller than stimulants; typically adjunctive rather than monotherapy
  • +1 more
Viloxazine
Consider when
  • Pediatric ADHD (ages 6–17) — FDA-approved ER formulation; newest non-stimulant option with novel NRI + 5-HT modulation
  • ADHD with comorbid anxiety — serotonergic component may benefit comorbid anxiety; non-stimulant avoids anxiety worsening
  • Stimulant refusal or contraindication — non-scheduled alternative; no abuse potential
  • Rapid onset for a non-stimulant — clinical improvement seen within 1 week in trials; faster than atomoxetine's 4–6 weeks
  • +1 more
Consider an alternative when
  • Adult ADHD — FDA-approved only for children/adolescents 6–17; adult use is off-label with limited data
  • Somnolence is problematic — 11–16% across doses; may impair daytime function
  • Nausea sensitivity — GI side effects (nausea, vomiting, decreased appetite) are common dose-dependent AEs
  • Long-term data needed — newest agent with limited long-term safety and efficacy data compared to atomoxetine or guanfacine
  • +1 more
Clonidine
Consider when
  • ADHD adjunct to stimulants — Kapvay ER FDA-approved as ADHD adjunct and monotherapy (ages 6–17); proven add-on to stimulants
  • ADHD with comorbid tics — α2 agonism benefits both ADHD and tic symptoms; CADDRA-recommended for this combination
  • Stimulant-induced insomnia — bedtime dosing leverages sedative effect to improve sleep onset in stimulant-treated patients
  • ADHD with comorbid aggression or emotional dysregulation — calms hyperarousal; evidence for reducing aggressive behavior
  • +1 more
Consider an alternative when
  • Hypotension risk — more hypotensive than guanfacine; α2A/B/C non-selective means broader cardiovascular effects
  • Abrupt discontinuation likely — rebound hypertension more severe than guanfacine; mandatory gradual taper
  • Daytime sedation problematic — more sedating than guanfacine; less α2A-selective means more non-specific sedation
  • Maximum ADHD efficacy needed — effect sizes smaller than stimulants and possibly guanfacine for core ADHD symptoms
  • +1 more
Drug-Drug Interactions2 moderate

Educational reference only. Interactions are extracted from FDA prescribing information and DDInter 2.0. Always verify with institutional pharmacy systems before clinical decisions.

Efficacy & Acceptability (2 axes)— NMA efficacy & discontinuation data (not side effects)
Axis
Atomoxetine
Guanfacine
Viloxazine
Clonidine
📊 Efficacy (response rates)
ADHDEfficacy
🛡️ Acceptability (all-cause discontinuation)
GeneralAcceptability
Axis
Atomoxetine
NRI
Guanfacine
alpha-2-agonist
Viloxazine
SNRI-ADHD
Clonidine
alpha-2-agonist
Boxed Warnings
Suicidality (boxed warning)
Mania / hypomania induction
Abuse / addiction liability
CNS
Sedation / somnolence
Activation / insomnia
Seizure risk
Other neurologic effects
Dizziness
Fatigue / lethargy
Metabolic
Weight gain
Appetite suppression / anorexia
Autonomic
Urinary retention / hesitancy
Sweating
Angle-closure glaucoma
Cardiac
QTc prolongation
Cardiac conduction / AV block
Serious CV / sudden death (ADHD labeled axis)
Blood pressure elevation
Heart rate / tachycardia
GI
Nausea / GI (general)
Hepatic
Liver enzymes / hepatotoxicity
Dermatologic
Rash (including SJS/TEN, pruritus, hypersensitivity)
Sexual
Sexual dysfunction
GU
Priapism
Discontinuation
Withdrawal / discontinuation
Rebound hypertension (alpha2-agonist discontinuation)
Interactions
MAOI co-administration contraindication
CYP interactions / DDI profile
Safety
Overdose toxicity
Pregnancy
Lactation / breastfeeding safety
Drug-specific / distinctive axes
Pheochromocytoma (§4.4 CI)
only in Atomoxetine

Safety: Every rating traces to a verbatim primary-source quote. Click any cell to audit. Stubs are disabled until calibrated. This tool surfaces published evidence — it does not replace clinical judgment.